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red and inflamed in appearance. Later, the abscess--for abscess it is--discharges its contents, the opening is explored, and we find that in extent it is not confined to the coronary region, but that it is deep enough to constitute a true sub-horny quittor. This discharge of the abscess contents may take place at a well-defined spot on the coronet, or it may ooze out at the junction of the wall with the skin. In appearance the discharged pus varies. When the softer structures only are attacked it is thick, and yellow or white in colour; when bone is involved it is ichorous; and when attacking the horn itself black or gray. It may or may not be extremely foetid, and often it is mingled with blood. When evidence of a previous opening upon the coronet is plain, then it is not considered wise to attempt a paring of the sole. Instead, poulticing is at once resorted to, to induce the discharge of the pus through its original channel. Once this has occurred a fistulous wound remains, which is open for treatment upon one or other of the lines we shall afterwards indicate. COMPLICATIONS--_(a) Necrosis of the Lateral Cartilage_.--This is the so-called 'cartilaginous quittor' of other writers. In all probability it is the condition generally understood when the word 'quittor' is used by one practitioner to the other. Its tendency to keep the disease existing in a chronic form renders it of grave importance, and for that reason we give it first mention among the complications. It may occur as a sequel either of cutaneous or of sub-horny quittor, and may result either from actual wounding and infection of the cartilage, or from an attack on it of septic matter originating elsewhere. Unless there has been discovered a fistula, which on probing is seen to lead direct to the position in which we know the cartilage to be, we know of no precise means by which the existence of this condition may be diagnosed. When free from other complications, the horse with his foot in this state may travel fairly sound. This is so when the necrosis is situate in the posterior half of the cartilage, in which case the irritation set up by the disease is confined to the comparatively non-sensitive tissues of the cartilage itself and the fibrous mass of the plantar cushion. When attacking the anterior half of the cartilage, the close contiguity of the joint renders the disease of a more serious nature. It is then that we have acute pain, and wit
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