nt appear,
indicating the formation of matter, it may be opened with a sharp lancet
and the wound treated daily with a solution of a teaspoonful of carbolic
acid in a half pint of water. Usually, however, when the inflammation has
proceeded to this extent, the gland will be ruined for purposes of
procreation and must be cut out. (See "Castration," p. 299.)
INFLAMMATION OF THE SHEATH.
While this may occur in bulls from infection during copulation and from
bruises, blows, and other mechanical injuries, the condition is more common
in the ox in connection with the comparative inactivity of the parts. The
sheath has a very small external opening, the mucous membrane of which is
studded with sebaceous glands secreting a thick, unctuous matter of a
strong, heavy odor. Behind this orifice is a distinct pouch, in which this
unctuous matter is liable to accumulate when the penis is habitually drawn
back. Moreover, the sheath has two muscles (protractors) which lengthen it,
passing into it from the region of the navel, and two (retractors) that
shorten it, passing into it from the lower surface of the pelvic bones
above. (Pl. IX, fig. 2.) The protractors keep the sheath stretched, so that
it habitually covers the penis, while the retractors shorten it up in the
act of service, so that the penis can project to its full extent. In stud
bulls the frequent protrusion of the erect and enlarged penis and the
retraction and dilation of the opening of the sheath serve to empty the
pouch and prevent any accumulation of sebaceous matter or urine. In the ox,
on the other hand, the undeveloped and inactive penis is usually drawn back
so as to leave the anterior preputial pouch empty, so that the sebaceous
matter has space to accumulate and is never expelled by the active
retraction of the sheath and protrusion of the erect penis in service.
Again, the ox rarely protrudes the tip of the penis in urination, the urine
is discharged into the preputial pouch and lodges and decomposes there, so
that there is a great liability to the precipitation of its earthy salts in
the form of gravel. The decomposing ammoniacal urine, the gritty crystals
precipitated from it, and the fetid, rancid, sebaceous matter set up
inflammation in the delicate mucous membrane lining the passage. The
membrane is thickened, reddened, rendered friable, and ultimately
ulcerated, and the now narrowed sheath is blocked by the increasing mass of
sebaceous and urinous mater
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