FREE BOOKS

Author's List




PREV.   NEXT  
|<   215   216   217   218   219   220   221   222   223   224   225   226   227   228   229   230   231   232   233   234   235   236   237   238   239  
240   241   242   243   244   245   246   247   248   249   250   251   252   253   254   255   256   257   258   259   260   261   262   263   264   >>   >|  
n, progressive bleeding was seldom observed. Again, when the wounds were explored, the amount of blood, although considerable, was usually not more than sufficed to fill up the space consequent on the loss of brain tissue. This was especially striking when large venous sinuses, as the superior longitudinal, were involved in the injury. None the less, haemorrhage at the base of the brain was, I believe, responsible for early death in many of the severe cases, especially when the wounds were near the lower regions of the skull. Escape of cerebro-spinal fluid was not so prominent a feature as might have been expected, considering how freely the arachnoid space was opened up in many cases. I think this was usually checked by early coagulation of the blood, and later by adhesions. It must be remembered also that extensive wounds were most common on the vertex, or at any rate over the convex surface of the brain, while fractures of the middle fossa were usually rapidly fatal. _Concussion._--Cases exhibiting symptoms of pure uncomplicated concussion were distinctly rare, as would be expected from the mechanism of the injuries. On the other hand, symptoms of concussion formed the dominant feature of all severe cases. The symptoms in many instances consisted in great part in transitory signs of the so-called 'radiation' type, such as are seen in destructive lesions where the signs of nervous damage rapidly tend to diminish and localise themselves. As to the causation of the 'radiation' symptoms, it is difficult to discriminate the effects of neighbouring parenchymatous haemorrhages from those of local vibratory concussion of the nervous tissue. The local character of the signs seems, however, to point to causation by molecular disturbance, resulting from the conduction of forcible mechanical vibration to the brain tissue rather than to upset in the intra-cranial pressure. Again the limited nature of the paralysis observed, sharply defines it from the general loss of power accompanying ordinary cases of concussion of the brain. The similarity of the phenomena to those described in other parts of the body under the heading of 'local shock' is sufficiently obvious. The following instance well exemplifies the condition in question: (55) Wounded at Spion Kop. A scalp wound 3 inches in length crossed the left parietal bone nearly transversely, starting 1-1/2 and ending 2 inches from the median line: the cen
PREV.   NEXT  
|<   215   216   217   218   219   220   221   222   223   224   225   226   227   228   229   230   231   232   233   234   235   236   237   238   239  
240   241   242   243   244   245   246   247   248   249   250   251   252   253   254   255   256   257   258   259   260   261   262   263   264   >>   >|  



Top keywords:

concussion

 

symptoms

 

tissue

 
wounds
 

observed

 
nervous
 

inches

 

causation

 

rapidly

 
feature

expected

 

severe

 

radiation

 

called

 

resulting

 

forcible

 

mechanical

 
disturbance
 
vibration
 
molecular

conduction

 

haemorrhages

 
localise
 

destructive

 

diminish

 

damage

 

lesions

 
cranial
 

neighbouring

 

parenchymatous


vibratory

 

effects

 

discriminate

 

difficult

 

character

 

phenomena

 

length

 
question
 

Wounded

 
crossed

ending

 

median

 

starting

 

transversely

 

parietal

 

condition

 

exemplifies

 

accompanying

 

ordinary

 

similarity