y acute, yet there is less
manifestation of pain than in arthritis.

It is only in structures such as the bursa intertubercularis or in the
sheath of the deep digital flexor that an inflammation causes much pain
and is apt to result in permanent lameness. This is due to the peculiar
character of the function of such structures.

An acute inflammation of a small bursa may even result in the
destruction of such synovial apparatus without serious inconvenience to
the subject, either at the time of destruction or thereafter.
Obliteration of the superficial bursa over the summit of the os calcis
is not likely to cause serious inconvenience or distress to the subject
unless it be due to an infected wound. Even then, with reasonably good
care given the animal, recovery is almost certain. Complete return of
function of the member and cessation of lameness takes place within a
few weeks in the average case.

Where an infectious synovitis involves a structure such as the sheath of
the tendon of the deep digital flexor (perforans) the condition is grave
and because of the location of this theca the prognosis is not much more
favorable than in an articular synovitis.

Inflammation of bursae and thecae may be classified on a chronological
basis with propriety because the duration of such affections, in many
cases, materially modifies the result. A chronic inflammatory
involvement of a theca through which an important tendon plays may cause
adhesions to form. Or there may occur erosions of the parts with
eventual hypertrophy and loss of function, partial or complete.

However, in general practice a classification on an etiological basis
is probably more practical and we shall consider inflammation of bursae
and thecae as _infectious_ and _noninfectious_.

_Infectious_ bursitis and thecitis is usually the result of direct
introduction of septic material into the synovial structure by means of
injuries. Infection by contiguous extension occurs and also metastatic
involvement is met with occasionally.

The noninfectious inflammation of bursae and thecae usually result from
contusions or strains and generally run their course without becoming
infective in character, where vitality and resistance of the subject are
normal.

In a general way, inflammation and other affections of bursae and thecae
are considered very similar to like affections of joints.

AFFECTIONS OF MUSCLES AND TENDONS.

Muscles and tendons having to do wit